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Primary Hyperoxaluria type 1 (PH) is a genetic disorder resulting from the hyperproduction of endogenous oxalate, caused by mutations in the AGXT gene. The disease results in increased serum and urinary levels of calcium oxalate, and consequent tissue deposit of oxalate crystals, lending to a progressive decline in renal function, and systemic oxalosis involving the kidneys, and other organs, including the heart. The present report describes a patient with PH1 oxalosis with postmortem analysis.
Molin et al. (Mon,) studied this question.