Intrinsic atrial vulnerability, rather than the accessory pathway, is a key mechanism for the genesis of paroxysmal atrial fibrillation in Wolff-Parkinson-White syndrome.
What are the mechanisms for the genesis of paroxysmal atrial fibrillation in the Wolff-Parkinson-White syndrome?
This review analyzes available data to determine whether paroxysmal atrial fibrillation in WPW syndrome is driven by intrinsic atrial vulnerability or the electrophysiological properties of the accessory pathway.
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Atrial vulnerability has been studied performing an atrial endocardial catheter mapping and analysing abnormal atrial electrograms. Other studies evaluated atrial refractoriness and intraatrial conduction times, suggesting an intrinsic atrial vulnerability as the mechanism of PAF and considering the AP as an innocent bystander. It is our intention to analyse the available data on this particular and interesting topic since AF has a singular prognostic significance in patients with the WPW syndrome, and its incidence is unusually high in the absence of any clinical evidence of cardiac organic disease.
Centurion et al. (Thu,) reported a other. Intrinsic atrial vulnerability, rather than the accessory pathway, is a key mechanism for the genesis of paroxysmal atrial fibrillation in Wolff-Parkinson-White syndrome.
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