The CD36 rs1761667 polymorphism may modulate CD36 transcription and is potentially linked to bladder cancer pathways, requiring further validation.
These results indicate that rs1761667 may modulate CD36 transcription in a genotype-dependent manner, particularly in the disease context. Overall, our findings point to a potential biological connection between inherited CD36 variation and bladder cancer-related pathways, underscoring the need for further validation in tumor tissues.
Păvălean et al. (Wed,) studied this question.