Increasing expression of presenilin 1, β-catenin, and p-PTEN and its regulatory roles on cell invasion in gastric cancer

Key Points

• Cell invasion in gastric cancer is regulated by presenilin 1 and beta-catenin expression levels, which are positively correlated.
• Increased expression of presenilin 1 correlates with elevated levels of p-PTEN and beta-catenin, indicating a potential pathway for tumor growth.
• This study employs correlation analysis to explore the interaction between presenilin 1, p-PTEN, and beta-catenin in gastric cancer cells.
• Findings suggest targeting these molecular pathways may help prevent gastric cancer progression and tumorigenesis.