April 28, 2006

Gut-Lymph Hypothesis of Systemic Inflammatory Response Syndrome/Multiple-Organ Dysfunction Syndrome: Validating Studies in a Porcine Model

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Abstract

This large animal model validates rodent studies showing that the shock-injured gut releases biologically active factors into the mesenteric lymph and these factors activate neutrophils and injure endothelial cells.

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Senthil et al. (Fri,) studied this question.

synapsesocial.com/papers/69de7d7b7ed287395e559256 https://doi.org/https://doi.org/10.1097/01.ta.0000215500.00018.47

Also Consider

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1TRAUMA-HEMORRHAGIC SHOCK MESENTERIC LYMPH FROM RAT CONTAINS A MODIFIED FORM OF ALBUMIN THAT IS IMPLICATED IN ENDOTHELIAL CELL TOXICITY2005 · 61 citations
2Factors in Intestinal Lymph after Shock Increase Neutrophil Adhesion Molecule Expression and Pulmonary Leukosequestration2003 · 38 citations
3POST-HEMORRHAGIC SHOCK MESENTERIC LYMPH IS CYTOTOXIC TO ENDOTHELIAL CELLS AND ACTIVATES NEUTROPHILS1998 · 122 citations
4The Role of Intestinal Barrier Failure and Bacterial Translocation in the Development of Systemic Infection and Multiple Organ Failure1990 · 487 citations
5THE EFFECTS OF ISCHEMIA AND ISCHEMIA-REPERFUSION ON BACTERIAL TRANSLOCATION, LIPID PEROXIDATION, AND GUT HISTOLOGY1992 · 36 citations

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