The provided abstract contains only background information on chronic inflammation and aging, and does not report any quantitative results regarding adiponectin, inflammatory markers, or survival.
Cohort
T HE association between chronic inflammation and aging- related decline is well documented (1). According to the molecular inflammation hypothesis, aging is accompanied by a diminished capacity for maintenance of cellular redox balance (2). The resulting accumulation of reactive oxidative species leads to chronic upregulation of inflammatory molecules, which act both as markers of heightened oxidative stress and as direct mediators of further homeostatic dysregulation across organ systems (2). Such chronically elevated levels of inflammatory markers are a common feature not just of reduced survival in older cohorts but also of a variety of aging-associated diseases (1,2).
Kizer et al. (Thu,) conducted a cohort in Aging. Adiponectin and inflammatory markers was evaluated on Survival. The provided abstract contains only background information on chronic inflammation and aging, and does not report any quantitative results regarding adiponectin, inflammatory markers, or survival.