Arrhythmogenic calmodulin mutations share a common defect characterized by diminished inhibition and facilitated activation of RyR2-mediated calcium release.
Absolute Event Rate: 0% vs 0%
release is a common defect of arrhythmogenic CaM mutations.
Søndergaard et al. (Sun,) reported a other. Arrhythmogenic calmodulin mutations share a common defect characterized by diminished inhibition and facilitated activation of RyR2-mediated calcium release.