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January 20, 2023CureusOpen Access

Thyrotoxicosis-Induced Cardiomyopathy With Systolic Dysfunction

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Why the study?

Thyrotoxicosis-induced dilated cardiomyopathy is a rare but potentially life-threatening complication that can cause irreversible changes in cardiac structure and function if left untreated.

Population

One 39-year-old patient with thyrotoxicosis-induced acute heart failure

Design

Case report

Key result

Methimazole, metoprolol, and sacubitril/valsartan therapy successfully reversed thyrotoxicosis-induced dilated cardiomyopathy, improving the patient's ejection fraction from 36% to 56%.

Authors

YKYusuf KhalilMDMichael D DubeLWLarry Woods

Discussion

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Overview

May support combined antithyroid and GDMT for thyrotoxic cardiomyopathy; leaves open generalizability pending controlled trials.

Study Design

Type

Case Report (n=1)

Multicenter

No

Structured PICO

P
Population
1 39-year-old African American male with a history of tobacco, cannabinoid, and fentanyl use, presenting with thyrotoxicosis-induced acute heart failure (LVEF 36%) secondary to Grave's disease.
I
Intervention
Methimazole 20 mg oral twice daily, metoprolol 50 mg oral daily, and sacubitril/valsartan 49/51 mg oral twice daily.
O
Outcome
Reversal of cardiac dysfunction (ejection fraction) and return to baseline functional status at 6 weeks.surrogate

Prompt intensive treatment of thyrotoxicosis-induced cardiomyopathy with anti-thyroid and heart failure medications can rapidly reverse severe left ventricular systolic dysfunction.

Limitations

  • Reversibility of hyperthyroidism-induced ventricular dysfunction may vary among patients
  • Need for more research to better understand the underlying mechanisms and long-term outcomes of hyperthyroidism-induced cardiomyopathy

Cite This Study

Khalil et al. (2023) conducted a case report in Thyrotoxicosis-induced dilated cardiomyopathy (n=1). Methimazole, metoprolol, and sacubitril/valsartan was evaluated on Left ventricular ejection fraction. Methimazole, metoprolol, and sacubitril/valsartan therapy successfully reversed thyrotoxicosis-induced dilated cardiomyopathy, improving the patient's ejection fraction from 36% to 56%.

synapsesocial.com/papers/6a0961d1a9b58856443410behttps://doi.org/10.7759/cureus.33988
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