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Glutamate acts on acid-sensing ion channels to worsen ischaemic brain injury | Synapse

July 10, 2024Open Access

Glutamate acts on acid-sensing ion channels to worsen ischaemic brain injury

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Resumen

Glutamate is traditionally viewed as the first messenger to activate NMDAR (N-methyl-D-aspartate receptor)-dependent cell death pathways in stroke

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Cite This Study

Lai et al. (Wed,) studied this question.

synapsesocial.com/papers/68e60ad6b6db64358759e8d6 https://doi.org/https://doi.org/10.1038/s41586-024-07684-7

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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Hit identification against peptidyl-prolyl isomerase of Theileria annulata by combined virtual high-throughput screening and molecular dynamics simulation approach2020 · 4 citations
2Alternative splicing of GluN1 gates glycine site–dependent nonionotropic signaling by NMDAR receptors2021 · 21 citations
3Elevation of the Extracellular Concentrations of Glutamate and Aspartate in Rat Hippocampus During Transient Cerebral Ischemia Monitored by Intracerebral Microdialysis1984 · 2,879 citations
4Gating mechanisms of acid-sensing ion channels2018 · 246 citations
5Synaptic versus extrasynaptic NMDA receptor signalling: implications for neurodegenerative disorders2010 · 1,609 citations