N-EVs delivering miR-16-5p reduce myocardial injury by suppressing CYP1B1-mediated PANoptosis, offering therapeutic potential for acute myocardial infarction.
Do N-EVs delivering miR-16-5p mitigate myocardial injury in preclinical models of acute myocardial infarction?
N-EVs delivering miR-16-5p show therapeutic potential for acute myocardial infarction by suppressing CYP1B1-mediated PANoptosis and promoting cardiac repair.
Tasa de eventos absoluta: 0% vs 0%
N-EVs mitigate myocardial injury by delivering miR-16-5p to suppress CYP1B1-mediated PANoptosis, highlighting their role in intercellular communication and therapeutic potential for AMI. This study provides insights into EV-based strategies for cardiac repair and fibrosis prevention.
Zhang et al. (Mon,) reported a other. N-EVs delivering miR-16-5p reduce myocardial injury by suppressing CYP1B1-mediated PANoptosis, offering therapeutic potential for acute myocardial infarction.