Endoplasmic reticulum stress and the unfolded protein response in ischemic nephropathy: Pathogenic mechanisms and emerging therapeutic strategies

Puntos clave

• Endoplasmic reticulum stress contributes significantly to ischemic nephropathy.
• The unfolded protein response may regulate cell survival and death pathways in these conditions.
• Assessment using key biomarkers includes insights into pathogenic mechanisms involved in ischemic nephropathy.
• This highlights the need for further exploration of novel therapeutic strategies for better patient outcomes.