Correction: Reciprocal regulation of oxidative stress and mitochondrial fission augments parvalbumin downregulation through CDK5-DRP1- and GPx1-NF-κB signaling pathways | Synapse
March 3, 2026Open Access
Correction: Reciprocal regulation of oxidative stress and mitochondrial fission augments parvalbumin downregulation through CDK5-DRP1- and GPx1-NF-κB signaling pathways
Puntos clave
Parvalbumin downregulation is enhanced through oxidative stress and mitochondrial fission pathways.
Key metrics indicate a significant involvement of cdk5 and drp1 in this process, leading to altered cell function.
Observational analysis examines the relationship between oxidative stress and signaling pathways affecting parvalbumin.
Understanding this reciprocal regulation may inform strategies for cellular health interventions.