Deoxynivalenol-induced apoptosis and IL-1β modulation by fosfomycin in porcine immune and human intestinal epithelial cells

Puntos clave

• Fosfomycin shows potential to reduce apoptosis in porcine immune cells exposed to deoxynivalenol, impacting cell survival.
• IL-1β levels significantly decrease with fosfomycin treatment, indicating immune response modulation.
• Analysis of intestinal epithelial cells demonstrates protective effects of fosfomycin against deoxynivalenol toxicity.
• Finding suggests that fosfomycin may offer a novel approach to alleviate toxin-induced cellular damage.