RAD51 promotes osteogenic differentiation and inhibits DNA damage in osteoporosis though regulating cGAS-STING signaling pathway
Puntos clave
Osteogenic differentiation was significantly promoted by RAD51, enhancing bone formation.
DNA damage was inhibited through the regulation of the cGAS-STING signaling pathway by RAD51.
This assessment utilized various experimental methods to explore RAD51's functions in osteoporosis.
These findings highlight RAD51 as a potential target for new osteoporosis therapies, necessitating further research.
Resumen
RAD51 facilitated osteogenic differentiation and attenuated DNA damage in OP by modulating the cGAS-STING signaling pathway, offering a potential novel therapeutic target for OP treatment.