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March 3, 2026
Cancer-associated fibroblasts promote immune evasion in pancreatic cancer via miR-181b-5p/STING/LGALS1 pathway
SZ
Shaobo Zhang
YG
Yuanyuan Guo
HQ
Haoran Qi
Central South University
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Puntos clave
Immune evasion occurs via the mir-181b-5p/STING/LGALS1 pathway, establishing a critical mechanism of tumor progression.
Key findings include that cancer-associated fibroblasts significantly enhance immune evasion markers in pancreatic cancer samples.
Analysis of tumor microenvironments indicates a direct relationship between fibroblast activity and immune suppression.
These findings suggest that targeting this pathway may provide new therapeutic strategies in pancreatic cancer treatment.
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Cancer-associated fibroblasts promote immune evasion in pancreatic cancer via miR-181b-5p/STING/LGALS1 pathway | Synapse
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Zhang et al. (Wed,) studied this question.
synapsesocial.com/papers/69a761c3c6e9836116a2fd3b
https://doi.org/https://doi.org/10.1016/j.canlet.2026.218331