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Development of glomerulonephritis during anti-TNF-α therapy for rheumatoid arthritis | Synapse

April 19, 2005Open Access

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Development of glomerulonephritis during anti-TNF-α therapy for rheumatoid arthritis

MSMichael B. StokesColumbia University Irving Medical Center KFKirk FosterUniversity of Nebraska System GMGlen S. MarkowitzColumbia University Irving Medical Center

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Resumen

Rheumatoid arthritis patients receiving anti-TNFalpha agents may develop glomerulonephritis via the induction of rheumatoid arthritis-related nephropathy or de novo autoimmune disorders.

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Cite This Study

Stokes et al. (Tue,) studied this question.

synapsesocial.com/papers/69d6bcab41375cf86eed896a https://doi.org/https://doi.org/10.1093/ndt/gfh832

Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Compromised Function of Regulatory T Cells in Rheumatoid Arthritis and Reversal by Anti-TNFα Therapy2004 · 1,240 citations
2Newer immunmodulating drugs in rheumatoid arthritis may precipitate glomerulonephritis2001 · 6 citations
3Nephrotic syndrome as a complication of anti-TNFalpha in a patient with rheumatoid arthritis.2003 · 31 citations
4The american rheumatism association 1987 revised criteria for the classification of rheumatoid arthritis1988 · 19,952 citations
5Antineutrophil cytoplasmic antibody-associated necrotizing crescentic glomerulonephritis in a patient receiving treatment with etanercept for severe rheumatoid arthritis2004 · 52 citations