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January 30, 2009Open Access

Apoptotic Mechanisms After Cerebral Ischemia

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Resumen

Although the ischemic penumbra is under threat of infarction, it is potentially salvageable and thus represents an opportunity for therapeutic intervention.

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Cite This Study

Broughton et al. (Fri,) studied this question.

synapsesocial.com/papers/69deab951d9bba5129b0c8e1 https://doi.org/https://doi.org/10.1161/strokeaha.108.531632

Also Consider

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1Overexpression of BCL-2 in transgenic mice protects neurons from naturally occurring cell death and experimental ischemia1994 · 1,096 citations
2Early Detection of DNA Strand Breaks in the Brain After Transient Focal Ischemia: Implications for the Role of DNA Damage in Apoptosis and Neuronal Cell Death1997 · 315 citations
3Caspase-Independent Programmed Cell Death Following Ischemic Stroke2008 · 84 citations
4Overexpression of Copper/Zinc Superoxide Dismutase in Transgenic Mice Protects against Neuronal Cell Death after Transient Focal Ischemia by Blocking Activation of the Bad Cell Death Signaling Pathway2003 · 121 citations
5Neuron-specific transgene expression of Bcl-XL but not Bcl-2 genes reduced lesion size after permanent middle cerebral artery occlusion in mice1999 · 95 citations

Apoptotic Mechanisms After Cerebral Ischemia | Synapse