INTRODUCTION: (henbane) contains tropane alkaloids responsible for antimuscarinic toxicity. This study aims to evaluate the clinical characteristics, management, and outcomes of pediatric henbane poisoning cases and to highlight prevention strategies. METHODS: considered significant. RESULTS: 0.031). No patients received physostigmine therapy. All patients recovered fully. DISCUSSION: Henbane poisoning in children causes neuropsychiatric and systemic signs of antimuscarinic toxicity, mainly agitation, hallucinations, and sinus tachycardia. Severe cases may lead to seizures and acute kidney injury, possibly due to reduced renal perfusion. CONCLUSION: Severe henbane poisoning in children requires sedation, can cause seizures, and may lead to acute kidney injury. Tachycardia is a potential indicator of renal injury.
Terzi et al. (Wed,) studied this question.