) to both porcine and murine upper respiratory tract epithelium. This study reveals a novel function of the autolysin Atl in promoting SS2 adhesion to the upper respiratory tract epithelium, which elucidates the underlying molecular mechanism and identifies the key functional domains involved. These findings offer new insights into autolysin biology and provide a theoretical basis for developing anti-adhesion strategies against streptococcal infections in pigs.
Liu et al. (Wed,) studied this question.