Plumbagin Induces Cell Cycle Arrest and Autophagy and Suppresses Epithelial to Mesenchymal Transition Involving PI3K/Akt/mTOR-Mediated Pathway in Human Pancreatic Cancer Cells Retraction

Puntos clave

• To examine how plumbagin influences cell cycle regulation and autophagy in pancreatic cancer cells through specific molecular pathways.
• Investigated the role of plumbagin in cellular processes in pancreatic cancer cells.
• Analyzed effects on the PI3K/Akt/mTOR signaling pathway.
• Studied the impact on epithelial to mesenchymal transition.
• The article was retracted, indicating issues with the findings, specifically regarding plumbagin's effects on cellular processes.