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Reply to ‘Mechanistic and systemic perspectives on TWEAK in pemphigus vulgaris’ | Synapse

February 13, 2026

Reply to ‘Mechanistic and systemic perspectives on TWEAK in pemphigus vulgaris’

Key Points

To address the insights presented on TWEAK's role in pemphigus vulgaris and its underlying mechanisms.
Response to previously published perspectives
Review of mechanistic insights
Discussion of systemic implications
Clarification of the mechanistic role of TWEAK
Emphasis on the immune response involved
Highlighting the inflammation associated with pemphigus vulgaris

Abstract

Original author’s response to ‘Mechanistic and systemic perspectives on TWEAK in pemphigus vulgaris’

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Cite This Study

Peng et al. (Wed,) studied this question.

synapsesocial.com/papers/698ebf4385a1ff6a9301680c https://doi.org/https://doi.org/10.1093/bjd/ljag056

Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Effect of opioid receptor antagonist on mitigating tumor necrosis factor-like weak inducer of apoptosis (TWEAK)-induced apoptolysis in pemphigus pathogenesis2024 · 11 citations
2Upadacitinib leading to resolution of refractory mucosal erosions in a case of pemphigus vulgaris2024 · 7 citations
3Single-cell sequencing reveals distinct immune cell features in cutaneous lesions of pemphigus vulgaris and bullous pemphigoid2024 · 17 citations
4Microenvironmental network of clonal CXCL13+CD4+ T cells and Tregs in pemphigus chronic blisters2023 · 34 citations
5Tumour necrosis factor-like weak inducer of apoptosis (TWEAK) is associated with inflammation and disease severity in pemphigus2025 · 7 citations