This article tests whether reduced local dynamical capacity can serve as a general marker of unconsciousness across sleep, propofol sedation, and epilepsy. Using a shared state-space framework, it shows that low capacity is present across all three regimes, but transport capture and pathological deformation dissociate them. Deep sleep remains comparatively innovation-dominated, propofol shows mixed capacity effects, and epilepsy shows the clearest combined profile of capacity collapse, capture, and pathological lock-in.
Robin Langell (Thu,) studied this question.