This case report details the presentation, diagnostic workup, and management of 57-year-old paramilitary personnel who developed acute onset, left-sided lower motor neuron facial paralysis, diagnosed as Bell's palsy. The patient presented with sudden facial weakness, inability to close the left eye, deviation of mouth, and loss of taste, following a prodromal episode of sore throat and dry cough. Neurological examination confirmed idiopathic lower motor neuron facial nerve palsy, with no abnormalities detected on non-contrast CT head or relevant serologies. A comprehensive treatment approach was adopted incorporating acyclovir, prednisolone with tapering, vitamin B complex, intensive eye care, and physical therapy exercises. Notably, the patient exhibited prompt clinical improvement within 10 days, regaining facial strength, eye closure ability, and taste sensation. The discussion highlights prevailing theories in Bell’s palsy pathogenesis, particularly viral reactivation and nerve compression, and supports combination corticosteroid and antiviral therapy for severe cases. The importance of vigilant eye protection and rehabilitative therapy is emphasized to minimize corneal complications and synkinesis. Psychosocial impact is also acknowledged, advocating holistic, multidisciplinary follow-up to support patient recovery and emotional health. This report underscores that early diagnosis, integrated medical management, and tailored rehabilitative strategies are critical for optimal outcomes in Bell’s palsy, while ongoing psychosocial support addresses concerns over facial changes and functional impairments during recovery.
Saha et al. (Tue,) studied this question.