Marine-derived curdepsidone A inhibits HeLa cell proliferation by modulating ER stress and PERK/ATF4/CHOP pathway

Key Points

• Curdepsidone A effectively inhibits HeLa cell proliferation through modulation of ER stress pathways.
• Key evidence shows that treatment with curdepsidone A significantly reduces cell growth by affecting the PERK/ATF4/CHOP pathway.
• This analysis employs cell culture techniques to evaluate the effects of curdepsidone A on HeLa cells over several days.
• The findings highlight the need for further exploration of curdepsidone A's potential in cancer therapy strategies.