What question did this study set out to answer?

This research aims to understand how Nicotiana benthamiana differs from Arabidopsis in activating salicylic acid biosynthesis through TIR signaling.

March 14, 2026

The SAG101–NRG1 branch of TIR signaling activates phenylalanine‐derived SA biosynthesis in Nicotiana benthamiana

Key Points

This research aims to understand how Nicotiana benthamiana differs from Arabidopsis in activating salicylic acid biosynthesis through TIR signaling.
Examined the SAG101-NRG1 pathway of TIR signaling in Nicotiana benthamiana
Analyzed salicylic acid biosynthesis activation
Compared signaling mechanisms between Nicotiana benthamiana and Arabidopsis
Identified the SAG101–NRG1 branch as key in activating salicylic acid production
Demonstrated evolutionary differences in immune signaling between plants
Revealed that both species utilize different mechanisms to control salicylic acid biosynthesis.

Abstract

Unlike Arabidopsis, Nicotiana benthamiana uses the SENESCENCE ASSOCIATED GENE 101- N REQUIREMENT GENE 1 branch of TIR signaling to activate salicylic acid biosynthesis, illustrating evolutionary divergence in how conserved immune signaling modules are deployed to control salicylic acid production across plant lineages.

The SAG101–NRG1 branch of TIR signaling activates phenylalanine‐derived SA biosynthesis in Nicotiana benthamiana

Key Points

Abstract

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