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The results suggest that CXCR2 mediates the atherogenic activity of environmental pollutants, such as dioxins, and contributes to the development of atherosclerosis through the induction of a vascular inflammatory response by activating the AhR-signaling pathway.
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Dalei Wu
University of Science and Technology of China
Noriko Nishimura
Memorial Sloan Kettering Cancer Center
Victoria Kuo
National Institute for Environmental Studies
Arteriosclerosis Thrombosis and Vascular Biology
National Institute for Environmental Studies
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Wu et al. (Fri,) studied this question.
synapsesocial.com/papers/69dcf7cf0de68e8319e53c35 — DOI: https://doi.org/10.1161/atvbaha.110.220202