paracrine/autocrine mechanisms. In this review, we discuss recent clinical and experimental data regarding molecular mechanisms of FGF23's paracrine action on the heart with respect to pathological cardiac remodeling.

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Leifheit‐Nestler et al. (Mon,) studied this question.

synapsesocial.com/papers/6a03ad8a4c16e573a6f015cd https://doi.org/https://doi.org/10.3389/fendo.2018.00278

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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Extrarenal effects of FGF232016 · 45 citations
2Fibroblast growth factor 23 is associated with left ventricular hypertrophy, not with uremic vasculopathy in peritoneal dialysis patients2016 · 38 citations
3Immune Cell and Other Noncardiomyocyte Regulation of Cardiac Hypertrophy and Remodeling2015 · 332 citations
4Loss of Klotho Contributes to Kidney Injury by Derepression of Wnt/β-Catenin Signaling2013 · 372 citations
5The role of fibroblast growth factor 23 and Klotho in uremic cardiomyopathy2016 · 59 citations

Paracrine Effects of FGF23 on the Heart | Synapse