April 22, 2026Open Access

HERV2365 upregulates FGF1 expression by sponging miR-326 to promote the progression of intrahepatic cholangiocarcinoma

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Abstract

. Mechanistically, HERV2365 sponged miR-326, thereby derepressing fibroblast growth factor 1 (FGF1) and promoting tumor growth. Collectively, our findings identified HERV2365 as an unfavorable prognostic marker by promoting ICC progression through regulating the miR-326/FGF1 axis.

HERV2365 upregulates FGF1 expression by sponging miR-326 to promote the progression of intrahepatic cholangiocarcinoma

Key Points

Abstract

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