Key result
Unrecognized wild-type transthyretin amyloidosis affects up to ~15% of older adults with heart failure.
Why the study?
Transthyretin amyloid cardiomyopathy is an under-recognized cause of heart failure in older adults with limited diagnostic certainty and evolving treatment options.
Transthyretin amyloid cardiomyopathy is an under-recognized cause of heart failure in older adults that can now be diagnosed noninvasively and treated with emerging pharmaceutical therapies.
Transthyretin amyloid cardiomyopathy (ATTR-CM) is an under-recognized cause of heart failure (HF) in older adults, resulting from myocardial deposition of misfolded transthyretin (TTR) or pre-albumin. Characteristic patterns of echocardiography and cardiac magnetic resonance can strongly suggest the disease but are not diagnostic. The diagnosis can be made with noninvasive nuclear imaging when there is no evidence of a monoclonal protein. Amyloid fibril formation results from a destabilizing mutation in hereditary ATTR amyloidosis (hATTR) or from an aging-linked process in wild-type ATTR amyloidosis (wtATTR). Recent studies have suggested that up to 10% to 15% of older adults with HF may have unrecognized wtATTR. Associated features, including carpal tunnel syndrome and lumbar spinal stenosis, raise suspicion and may afford a means for early diagnosis. Previously treatable only by organ transplantation, pharmaceutical therapy that slows or halts ATTR-CM progression and favorably affects clinical outcomes is now available. Early recognition remains essential to afford the best treatment efficacy.
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Ruberg et al. (2019) conducted a review in Transthyretin amyloid cardiomyopathy (ATTR-CM). Up to 10% to 15% of older adults with heart failure may have unrecognized wild-type transthyretin amyloidosis, which can now be diagnosed noninvasively and treated with pharmaceutical therapy.
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