Key result
Newly developed chemical entities that increase the activity of I(Kr), I(Ks), and I(K1) are being evaluated as a potential novel anti-arrhythmic principle.
Why the study?
Does an increase in repolarization capacity through activation of I(Kr), I(Ks), and I(K1) constitute a new anti-arrhythmic principle?
Design
Review
Authors
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Identifies Kv11.1, Kv7.1 and Kir2.1 as primary phase 3 channels; leaves open whether boosting repolarization capacity yields new antiarrhythmic strategies.
Does an increase in repolarization capacity through activation of I(Kr), I(Ks), and I(K1) constitute a new anti-arrhythmic principle?
Pharmacological activation of cardiac potassium channels (I(Kr), I(Ks), I(K1)) to increase repolarization capacity is being explored as a novel anti-arrhythmic strategy.
Morten Grunnet (2010) conducted a review in Arrhythmias. Chemical entities increasing activity of I(Kr), I(Ks) and I(K1) was evaluated. Newly developed chemical entities that increase the activity of I(Kr), I(Ks), and I(K1) are being evaluated as a potential novel anti-arrhythmic principle.
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