Knowledge about the exact anastomotic pathways and structural changes in the coronary circulation during obliterative coronary artery disease remains sketchy.
Most investigators are agreed that frequent arterial anastomoses of relatively large size may be demonstrated in the coronary circulation in the presence of obliterative coronary artery disease. Evidence has been presented which shows that the enlarged anastomotic channels found in disease may in fact be derived from pre-existing, pre-capillary communications (Fulton, 1960, 1963a,b). Although there is a considerable body of information concerning the effects of coronary artery ligation on collateral blood flow in the experimental animal (Gregg, 1950) and on the increased incidence of readily injectable intercoronary communication in the presence of coronary occlusion (Zoll, Wessler, and Blumgart, 1951a), knowledge about the exact anastomotic pathways concerned and detail of their structural change is still somewhat sketchy.
W. F. M. Fulton (Wed,) conducted a review in obliterative coronary artery disease. Knowledge about the exact anastomotic pathways and structural changes in the coronary circulation during obliterative coronary artery disease remains sketchy.