What is the clinical evidence from this study?

Study design: Other. Population: Hypertension and Normotension (n=26). Intervention: Resistance exercise vs. Control (rest). Primary outcome: Change in systolic and diastolic blood pressure at 30 to 60 minutes post-intervention (p=> 0.05).

July 1, 2014

Post‐resistance exercise hemodynamic and autonomic responses: Comparison between normotensive and hypertensive men

Key Result

A session of resistance exercise promoted post-exercise hypotension in both normotensive and hypertensive men, but the effect did not persist during the 24-hour ambulatory period.

PICO

Population

26 normotensive and hypertensive men who underwent control (rest) and resistance exercise sessions with 24-hour ambulatory blood pressure monitoring.

Intervention / Comparator

Resistance exercise vs Control (rest) (Seven exercises, three sets, 50% of one repetition maximum)

Primary Outcome

Change in systolic and diastolic blood pressure at 30 to 60 minutes post-intervention, p=> 0.05

Main Result

p-value: p=> 0.05

Limitations

The post-resistance exercise hypotension did not last during the ambulatory period, which limits its clinical relevance.

Abstract

To compare post-resistance exercise hypotension (PREH) and its mechanisms in normotensive and hypertensive individuals, 14 normotensives and 12 hypertensives underwent two experimental sessions: control (rest) and exercise (seven exercises, three sets, 50% of one repetition maximum). Hemodynamic and autonomic clinic measurements were taken before (Pre) and at two moments post-interventions (Post 1: between 30 and 60 min; Post 2: after 7 h). Ambulatory blood pressure (BP) was monitored for 24 h. At Post 1, exercise decreased systolic BP similarly in normotensives and hypertensives (-8 ± 2 vs -13 ± 2 mmHg, P > 0.05), whereas diastolic BP decreased more in hypertensives (-4 ± 1 vs -9 ± 1 mmHg, P 0.05). After exercise, heart rate (+13 ± 3 vs +13 ± 2 bpm) and its variability (low- to high-frequency components ratio, 1.9 ± 0.4 vs +1.4 ± 0.3) increased whereas stroke volume (-14 ± 5 vs -11 ± 5 mL) decreased similarly in normotensives and hypertensives (all, P > 0.05). At Post 2, all variables returned to pre-intervention, and ambulatory data were similar between sessions. Thus, a session of resistance exercise promoted PREH in normotensives and hypertensives. Although this PREH was greater in hypertensives, it did not last during the ambulatory period, which limits its clinical relevance. In addition, the mechanisms of PREH were similar in hypertensives and normotensives.

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