Emergent angiographic assessment and echocardiography revealed no anatomical or functional evidence of obstructive coronary disease in a patient with acute pancreatitis mimicking myocardial infarction.
Case Report (n=1)
Acute pancreatitis can mimic acute myocardial infarction with ST segment elevation and cardiac enzyme derangements in the absence of obstructive coronary disease, highlighting the utility of early angiography to avoid inappropriate thrombolysis.
The authors report the first emergent angiographic assessment of the coronaries with accompanying echocardiography in a 64-year-old man with dermatomyositis, who presented with ST segment elevation and cardiac specific enzyme derangements highly suggestive of myocardial infarction in the presence of acute pancreatitis. Both studies revealed no anatomical or functional evidence of obstructive coronary disease. Although the mechanism of electrocardiogram abnormalities found in acute pancreatitis remains to be elucidated fully, the authors propose a direct cardiac toxic effect by the pancreatic proteolytic enzymes to account for these changes and we recommend an angiographic approach as the first step to avoid the potentially lethal administration of thrombolytic therapy or potent anticoagulation.
Albrecht et al. (Thu,) conducted a case report in Acute pancreatitis mimicking myocardial infarction (n=1). Emergent angiographic assessment and echocardiography was evaluated on Anatomical or functional evidence of obstructive coronary disease. Emergent angiographic assessment and echocardiography revealed no anatomical or functional evidence of obstructive coronary disease in a patient with acute pancreatitis mimicking myocardial infarction.
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