Does sustained atrial tachycardia impair cellular contractility and Ca2+ handling in atrial cells?
Atrial cells (specific model not stated in abstract)
Sustained atrial tachycardia
Cellular contractility and Ca2+ handlingsurrogate
Sustained atrial tachycardia directly impairs cellular calcium handling and contractility, providing a mechanistic basis for tachycardia-induced atrial cardiomyopathy in atrial fibrillation.
Sustained atrial tachycardia causes important reductions in cellular contractility, in part by impairing cellular Ca2+ handling and decreasing systolic Ca2+ transients. These results provide direct evidence for the concept that AF induces atrial contractile dysfunction by causing a tachycardia-induced atrial cardiomyopathy.
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Hui Sun
Qingdao University
Rania Gaspo
University of Nueva Caceres
Normand Leblanc
University of Nevada, Reno
Circulation
McGill University
Université de Montréal
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Sun et al. (Tue,) studied this question.
synapsesocial.com/papers/69d9c72f1ad561c6736851bc — DOI: https://doi.org/10.1161/01.cir.98.7.719