Hypoxic stress induces IL-6 production in cardiac myocytes, highlighting a potential mechanism for myocardial dysfunction in ischemia-reperfusion injury.
Cardiac myocytes respond to hypoxic stress to augment the production of IL-6, and the IL-6 derived from cardiac myocytes may play an important role in the progression of myocardial dysfunction observed in cardiac ischemia-reperfusion injury.
Yamauchi‐Takihara et al. (Wed,) studied this question.
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