Does subcutaneous exenatide prevent IR-induced endothelial dysfunction in a human IR injury model?
Exenatide demonstrates protective effects against ischemia-reperfusion-induced endothelial dysfunction via K(ATP) channel activation in a human model.
The present study demonstrates that subcutaneous exenatide protects IR-induced endothelial dysfunction through opening of K(ATP) channels in human IR injury model.
Ha et al. (Fri,) studied this question.