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Development of glomerulonephritis during anti-TNF-α therapy for rheumatoid arthritis | Synapse

April 19, 2005Open Access

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Development of glomerulonephritis during anti-TNF-α therapy for rheumatoid arthritis

MSMichael B. StokesColumbia University Irving Medical Center KFKirk FosterUniversity of Nebraska System GMGlen S. MarkowitzColumbia University Irving Medical Center

Key Points

Key points are not available for this paper at this time.

Abstract

Rheumatoid arthritis patients receiving anti-TNFalpha agents may develop glomerulonephritis via the induction of rheumatoid arthritis-related nephropathy or de novo autoimmune disorders.

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Cite This Study

Stokes et al. (Tue,) studied this question.

synapsesocial.com/papers/69d6bcab41375cf86eed896a https://doi.org/https://doi.org/10.1093/ndt/gfh832

Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Chronic therapy with recombinant tumor necrosis factor-α in autoimmune NZB/NZW F1 mice1989 · 168 citations
2Newer immunomodulating drugs in rheumatoid arthritis may precipitate glomerulonephritis.2001 · 24 citations
3Newer immunmodulating drugs in rheumatoid arthritis may precipitate glomerulonephritis2001 · 6 citations
4Nephrotic syndrome as a complication of anti-TNFalpha in a patient with rheumatoid arthritis.2003 · 31 citations
5Revised Criteria for the Classification of Rheumatoid Arthritis1990 · 7,735 citations