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This review surveys the signals, intermediate events, and effectors that connect GC swelling and shrinking to environmental conditions. GCs swell when protons are released, hyperpolarizing the plasma membrane (shifting the membrane potential to more negative voltages). This hyperpolarization drives K+ entry down an electrical gradient through inward K+ channels. C1-influx also occurs, presumably via Cl-/H+ symport or Cl-/OH-antiport. The entering ions are stored primarily in the vacuole along with malate, which is synthesized during stomatal opening. As intracellular osmotica increase, GCs take up water, balancing their water potential with that of their surroundings, and they begin to swell. Differential cell wall reinforcement bends the swelling GCs outward and widens the pore between them (Willmer, 1983; Taiz and Zeiger, 1991).
Kearns et al. (Thu,) studied this question.
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