This editorial discusses the concept of ventricular syncope triggered by activation of sensory nerve endings in the ventricles, comparing it to carotid-sinus hypersensitivity.
Ventricular syncope
To those who think of the heart as merely a pump, the concept that an episode of syncope may be triggered by activation of sensory nerve endings in the ventricles may seem far-fetched. It should not. We accept the notion than an arterial "bulge" at the carotid bifurcation can cause syncope if compressed or massaged vigorously, and we recognize the syndrome of carotid-sinus syncope and carotid-sinus hypersensitivity as a common clinical entity.The patients described in the paper by Almquist et al.1 in this issue of the Journal may have a comparable syndrome — that of ventricular syncope, or hypersensitivity . . .
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François M. Abboud
General Cardiology
New England Journal of Medicine
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François M. Abboud (Thu,) conducted a editorial in Ventricular syncope. This editorial discusses the concept of ventricular syncope triggered by activation of sensory nerve endings in the ventricles, comparing it to carotid-sinus hypersensitivity.
synapsesocial.com/papers/6a07b57715d371b388386afa — DOI: https://doi.org/10.1056/nejm198902093200609