Dysfunctional adipose tissue activates the renin-angiotensin-aldosterone system, which drives obesity-associated hypertension via renal, central, and vascular mechanisms.
This review highlights the mechanistic links between adipose tissue dysfunction, RAAS overactivity, and the development of obesity-associated hypertension.
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Obese individuals frequently develop hypertension, which is for an important part attributable to renin-angiotensin-aldosterone system (RAAS) overactivity. This review summarizes preclinical and clinical evidence on the involvement of dysfunctional adipose tissue in RAAS activation and on the renal, central, and vascular mechanisms linking RAAS components to obesity-associated hypertension.
Schütten et al. (Thu,) reported a other. Dysfunctional adipose tissue activates the renin-angiotensin-aldosterone system, which drives obesity-associated hypertension via renal, central, and vascular mechanisms.
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