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mice. Our studies reveal that neutrophils use an inflammasome- and GSDMD-dependent mechanism to activate NETosis as a defense response against cytosolic bacteria.
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Kaiwen Chen
Mercedes Monteleone
Dave Boucher
Science Immunology
The University of Queensland
University of Lausanne
Max Planck Institute for Infection Biology
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Chen et al. (Fri,) studied this question.
www.synapsesocial.com/papers/69faf128ed76930a72df79bc — DOI: https://doi.org/10.1126/sciimmunol.aar6676