Ablation of class X myosin (Myo10) inhibited filopodia formation and myoblast fusion in vitro, and severely impaired postnatal muscle regeneration in mice.
Myo10-driven filopodia are essential for mammalian myoblast fusion and postnatal muscle regeneration.
Skeletal muscle fibers are multinucleated cellular giants formed by the fusion of mononuclear myoblasts. Several molecules involved in myoblast fusion have been discovered, and finger-like projections coincident with myoblast fusion have also been implicated in the fusion process. The role of these cellular projections in muscle cell fusion was investigated herein. We demonstrate that these projections are filopodia generated by class X myosin (Myo10), an unconventional myosin motor protein specialized for filopodia. We further show that Myo10 is highly expressed by differentiating myoblasts, and Myo10 ablation inhibits both filopodia formation and myoblast fusion in vitro. In vivo, Myo10 labels regenerating muscle fibers associated with Duchenne muscular dystrophy and acute muscle injury. In mice, conditional loss of Myo10 from muscle-resident stem cells, known as satellite cells, severely impairs postnatal muscle regeneration. Furthermore, the muscle fusion proteins Myomaker and Myomixer are detected in myoblast filopodia. These data demonstrate that Myo10-driven filopodia facilitate multinucleated mammalian muscle formation.
Hammers et al. (Tue,) conducted a other in Muscle regeneration and myoblast fusion. Myo10 ablation (knockdown/conditional knockout) vs. Wild-type or control shRNA was evaluated on Myoblast fusion and muscle regeneration. Ablation of class X myosin (Myo10) inhibited filopodia formation and myoblast fusion in vitro, and severely impaired postnatal muscle regeneration in mice.
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