Beyond Hemostasis: Platelet Innate Immune Interactions and Thromboinflammation

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Abstract

, TLT-1, CLEC-2 and Toll-like receptors (TLRs) as well as secretions from platelet granules on platelet-neutrophil aggregate and neutrophil extracellular trap (NET) formation in atherosclerosis, transfusion-related acute lung injury (TRALI) and COVID-19. Further, we will address platelet-monocyte and macrophage interactions during cancer metastasis, infection, sepsis and platelet clearance.

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Authors

Jonathan Mandel

Johannes Gutenberg University Mainz

Martina Casari

M. G. Stepanyan

Yerevan State University

Journals

International Journal of Molecular Sciences

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Institutions

Johannes Gutenberg University Mainz

Lomonosov Moscow State University

University Medical Center of the Johannes Gutenberg University Mainz

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Mandel et al. (Thu,) studied this question.

synapsesocial.com/papers/69f3ee89dc238f819779984d — DOI: https://doi.org/10.3390/ijms23073868

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Also consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

Caspases and their substrates· 2017 · 805 citations
Neutrophil arrest by LFA-1 activation· 2012 · 128 citations
Peripherally derived macrophages can engraft the brain independent of irradiation and maintain an identity distinct from microglia· 2018 · 414 citations
Dynamics of leukocyte-platelet adhesion in whole blood· 1991 · 327 citations
Neutrophil-Platelet Interactions as Novel Treatment Targets in Cardiovascular Disease· 2022 · 75 citations

Beyond Hemostasis: Platelet Innate Immune Interactions and Thromboinflammation

Key Points

Abstract

Citation Network

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Discussion

Authors

Journals

Actions

Institutions

References and Citations

Citation Network

Connected Papers

Discussion

Cite this study

Also consider

Also consider