Left ventricular pressure overload model
Cardiac fibroblast accumulation, ECM secretion, and extent of fibrosis
Identifies p53-dependent cell cycle control as a key mechanism regulating cardiac fibrosis in left ventricular pressure overload.
This study reveals a mechanism regulating cardiac fibroblast accumulation and ECM secretion, orchestrated in part by p53-dependent cell cycle control that governs the timing and extent of fibrosis in left ventricular pressure overload.
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Xiaoyi Liu
Fujian Medical University
Ryan M. Burke
Stanford Medicine
Janet K. Lighthouse
University of Rochester Medical Center
Circulation Research
University of Rochester
University of Rochester Medical Center
Pediatrics and Genetics
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Liu et al. (Thu,) studied this question.
synapsesocial.com/papers/69d758ea086f9d6299f30908 — DOI: https://doi.org/10.1161/circresaha.121.320324