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Glutamate acts on acid-sensing ion channels to worsen ischaemic brain injury | Synapse

July 10, 2024Open Access

Glutamate acts on acid-sensing ion channels to worsen ischaemic brain injury

Key Points

Key points are not available for this paper at this time.

Abstract

Glutamate is traditionally viewed as the first messenger to activate NMDAR (N-methyl-D-aspartate receptor)-dependent cell death pathways in stroke

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Cite This Study

Lai et al. (Wed,) studied this question.

synapsesocial.com/papers/68e60ad6b6db64358759e8d6 https://doi.org/https://doi.org/10.1038/s41586-024-07684-7

Also Consider

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1Global, Regional, and Country-Specific Lifetime Risks of Stroke, 1990 and 20162018 · 1,529 citations
2Mitochondrial Dysfunction Is a Primary Event in Glutamate Neurotoxicity1996 · 771 citations
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4Hit identification against peptidyl-prolyl isomerase of Theileria annulata by combined virtual high-throughput screening and molecular dynamics simulation approach2020 · 4 citations
5Synaptic versus extrasynaptic NMDA receptor signalling: implications for neurodegenerative disorders2010 · 1,608 citations