Aim: Polycystic ovary syndrome (PCOS) is a heterogeneous endocrine disorder affecting 5–10% of women of reproductive age and is increasingly recognized as a female-specific cardiometabolic condition. Although metabolic risk factors such as insulin resistance, dyslipidemia, and hypertension are well established in PCOS, their distribution across distinct phenotypes remains controversial. Material and Methods: In this retrospective study, 240 women diagnosed with PCOS based on the 2003 Rotterdam criteria and 116 healthy controls aged 18–42 years were evaluated. Patients with PCOS were categorized into four phenotypes: (i) hyperandrogenism + oligo/anovulation + polycystic ovaries (HA+OA+PCO), (ii) hyperandrogenism + oligo/anovulation (HA+OA), (iii) hyperandrogenism + polycystic ovaries (HA+PCO), and (iv) polycystic ovaries + oligo/anovulation (PCO+OA). Anthropometric measurements, hormonal profiles, lipid panels, glucose-insulin parameters, and HOMA-IR indices were compared between groups. Results: Compared with controls, women with PCOS had significantly higher triglycerides, fasting insulin, HOMA-IR, total testosterone, DHEAS, and LH/FSH ratio, while HDL cholesterol was lower (all p
Aksu et al. (Sat,) studied this question.