High testosterone levels in spontaneously hypertensive rats are associated with increased mean arterial pressure and significant regulation of 6,571 genes in the paraventricular nucleus.
Does testosterone manipulation alter mean arterial pressure and paraventricular nucleus gene expression in spontaneously hypertensive rats?
Testosterone levels correlate with mean arterial pressure and drive significant transcriptomic changes in the paraventricular nucleus of spontaneously hypertensive rats, highlighting a hormonal mechanism for sex differences in hypertension.
Absolute Event Rate: 0% vs 0%
Abstract Background Hypertension is a polygenic, complex disease that impacts men and women differently; whilst the incidence of high blood pressure (BP) is roughly equal over a lifetime, men typically are at higher risk of developing the disease earlier in life, before 50 years of age. There is adequate evidence that the brain is critical for the BP setpoint. The paraventricular nucleus (PVN) of the hypothalamus is an integrative structure that can influence not only neurohumoral responses to blood pressure changes, but also sympathetic drive. Here we manipulate the androgenic status of both male and female spontaneously hypertensive rats (SHRs) to determine how this changes gene expression within the PVN of these animals. Methods SHR (8-weeks old) were either sham-operated or orchiectomized, whereas all females were oophorectomized, half of which received 10 mg testosterone propionate subcutaneously. Mean arterial pressure (MAP) and testosterone (T) were measured by carotid cannulation and ELISA respectively. Sequencing was performed on hand-punched PVN sections and subjected to robust bioinformatic analysis. Results in total, 6,571 differentially regulated genes (DRGs) are regulated in the PVN of male and female rats. High T (endogenous or replaced) correlates with higher MAP in both sexes. Orchidectomy-induced T depletion resulted in the significant regulation of 5,104 genes, involved in thousands of biological roles, including ones related to hormone and sex-hormone signalling. In the female SHR, testosterone replacement in oophorectomized animals induced the regulation of 1,727 genes, sharing many biological functions with those in the high T males. We validated key genes by qPCR to determine false discovery rate. Conclusions T status in hypertensive rats correlates with MAP, and consistent changes in PVN transcriptome.
Paterson et al. (Thu,) reported a other. High testosterone levels in spontaneously hypertensive rats are associated with increased mean arterial pressure and significant regulation of 6,571 genes in the paraventricular nucleus.
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