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This work aims to provide a comprehensive index for the Lantern of Sulfur framework and its implications for physiological understanding.

April 30, 2026Open Access

Master Index — Lantern of Sulfur (v2026.19, April 2026)

Key Points

This work aims to provide a comprehensive index for the Lantern of Sulfur framework and its implications for physiological understanding.
Organized by layers: Structure, Mechanism, Coherence, and Application
Defined Directional Pressure Failure behavior and mechanisms
Formalized compensation patterns in system behavior
Versioned index to enhance navigation and continuity
Identified hyperchloremia as a key marker of system disruption
Established connections between physiological processes and system behavior
Provided structured navigation to clinical patterns and interventions

Abstract

This record serves as the authoritative master index and navigation layer for the Lantern of Sulfur (LoS) framework and the associated Directional Pressure Failure (DPF) Series. The Lantern of Sulfur (LoS) defines physiology as a coordinated system in which coherence and collapse emerge from the interaction of structure, control, state, and failure. It organizes system behavior across four layers: • Structure (Concept A): invariant geometry governing containment, timing, and directionality• Mechanism (Concept B): biological execution across metabolic, electrolyte, and regulatory systems• Coherence (Concept C): patterns that hold or fail under load across domains• Application / Translation: clinical entry points, system interpretation, and stabilization strategies The Directional Pressure Failure (DPF) Series extends this framework into execution-level behavior, defining how systems remain active but fail to resolve when directional control, exit integrity, or timing alignment is lost. It formalizes compensation patterns including RAAS activation, fluid misdistribution, and electrolyte instability. Mechanistic Continuity Notes connect LoS structure and DPF failure to established physiology, showing how bile signaling, renal execution, nitric oxide dynamics, and electrolyte regulation express the same underlying system behavior. What this index provides • A guided entry system, allowing readers to enter through recognition, structure, failure, or clinical pattern• A layered navigation architecture, organizing the corpus across Concept A, B, C, DPF, and bridges• A complete, version-controlled map of the LoS and DPF preprint series Core framing Hyperchloremia is used throughout as a central system signal—not as an isolated electrolyte abnormality, but as a marker of disrupted coordination across hydration, acid–base balance, hormonal signaling, and metabolic timing. The framework enables movement from:→ observation → pattern recognition → system interpretation → controlled intervention Versioning This record is versioned through Zenodo to preserve continuity across updates while maintaining a stable conceptual DOI. Each version reflects refinement of system structure, alignment of the corpus, and improved navigability. Author: Beth A. MartellAffiliation: Independent ResearcherORCID: 0009-0005-6448-9364License: CC BY 4.0

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