Based on the core lifespan formula L = Tavg × N and the full pathological system of SFL-CELL serial, this paper deduces the unified universal anti-aging and disease intervention logic under the Species Fixed Law framework. The core invariant of human life is that the total cellular renewal quota N is fixed at birth and cannot be increased through any external intervention; all effective long-term health maintenance strategies can only start from two directions: reducing the accumulation of irreversible ΔGd(-) structural damage, and stabilizing or restoring the average cell functional cycle Tavg. The two core intervention paths are sorted systematically: 1. Block exogenous and endogenous sources of ΔGd(-): reduce chronic toxin exposure, relieve persistent inflammatory circulation, avoid excessive tissue trauma, and cut off the continuous erosion of cell G₀ functional modules; 2. Repair damaged G₀ architecture of core functional cells represented by bone marrow hematopoietic stem cells: restore platelet repair capacity, neuron protein clearance function, cell proliferation control modules, etc., so as to raise the whole-body Tavg and slow down the consumption rate of fixed renewal quota N. Conventional medical intervention modes are divided into two categories for discrimination: symptomatic suppression therapy (glucocorticoids, immunosuppressants, tumor resection, protein plaque clearance) only eliminates secondary pathological manifestations, cannot repair damaged G₀ structure, and cannot improve Tavg fundamentally; root-targeted repair therapy (mesenchymal stem cell transplantation, PRP supplementation, nerve cell microenvironment regulation) can restore partial G₀ integrity, effectively lift the suppressed Tavg and delay lifespan atrophy. Combined with the unified numerical baseline N=10 for demonstration: long-term standardized intervention can maintain Tavg close to the healthy baseline of 8 years, locking the theoretical lifespan near 80 years; long-term unregulated damage accumulation will keep Tavg at a low level, leading to rapid depletion of renewal quota and premature aging or early onset of fatal chronic diseases. This paper forms a complete closed-loop application system of the SFL-LS quantitative model, which echoes the unified chronic disease repair decline spectrum in SFL-CELL-06, and provides quantitative evaluation criteria for judging the long-term efficacy of all clinical and health maintenance schemes.
FOO SENG ANG (Sun,) studied this question.