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March 3, 2026Nature Communications3 citationsOpen Access

Kynurenine mediates the chemotherapy-induced intestinal toxicity through modulation of gut microbiota

HXHongyu XieJYJingyi YangJWJinjie Wu

Key Points

  • Intestinal toxicity significantly increases with elevated kynurenine levels, especially following oxaliplatin treatment.
  • Serum L-kynurenine levels rise due to IFNγ-driven induction of indoleamine 2,3-dioxygenase 1 in myeloid cells.
  • Analysis using single-cell RNA sequencing confirms that myeloid cell-derived kynurenine worsens gut dysbiosis and toxicity.
  • Targeting kynurenine may offer a therapeutic approach to mitigate intestinal injury caused by chemotherapy.

Abstract

Chemotherapy-induced intestinal toxicity is a major dose-limiting complication, but the underlying mechanisms linking systemic metabolism to localized gut damage are poorly understood. Here we show that serum L-kynurenine, a tryptophan metabolite, is elevated in patients with severe oxaliplatin-induced intestinal toxicity. Accumulation of L-kynurenine is driven by IFNγ-mediated induction of indoleamine 2,3-dioxygenase 1 (IDO1) in myeloid cells. Using scRNA-seq and myeloid cell-specific knockout models, we confirm that myeloid cell-derived L-kynurenine exacerbates toxicity. Critically, L-kynurenine accumulation drives gut dysbiosis, characterized by the loss of Lactobacillus johnsonii, and subsequently activates the TNFα/JNK pathway, leading to intestinal epithelial apoptosis. Pharmacological inhibition or engineered reduction of L-kynurenine mitigates chemotherapy-induced intestinal injury. Our findings reveal an important role of L-kynurenine from myeloid cells in chemotherapy tolerance and propose its targeting as a potential therapeutic strategy.

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Cite This Study

Xie et al. (2026) studied this question.

synapsesocial.com/papers/69a75b2ec6e9836116a2209ehttps://doi.org/10.1038/s41467-026-68741-5
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